Specific Hibernation (SH) is the result of a pragmatic violation of the standard lexical meaning of the word "hibernation". This violation can be justified because it contributes an explanatory logic beyond the fact that "hibernation" contributes an H whereby to form a deservedly (and 'distinKtly') allusive acronym from the expressions, "Specific Hibernation imploring threats", or "perils or ordeals of SH imploring type".
I hope it is as didactically helpful for someone else as it was autodidactically for me to compare SH with all examples of with seasonal regularity instinctively prepared for and entered into states of dormancy (that is aestivation as well as hibernation). Such ways of coping with seasonally harsh climatic conditions may be referred to as General 'Hibernation' (GH).
In other words, "specific hibernation" got (happened) to be ÆPTly chosen because it fitted both a MAD-inspired acronym-building aim and because the expression provides an apt reference to the obvious fact that a relatively metabolically muted metabolism and a compromised (cancelled) consciousnessT is the common denominator between SH and GH.
Specific Hibernation (SH) is a concEPT that refers to presynaptic or postsynaptic inhibitory functions that most directly initiate and typically inertly maintain an absence of firing or a sufficiently low frequency of firing (with a correspondingly muted neuronal metabolism) of postsynaptic and downstream neurons of actention modules that are phylogenetically dedicated to as if by default "pay/focus actentions of distress-type.
So, in yet other ÆPTly explanatory words, SH refers to an indefinitely distress-delaying absence of stimulation of excitatory neurons downstream of sites of such inhibitory (SH inducing) neural control (with an ASSS)—a signaling prepared or led up to by sensory detection and registration of environmental distressors of a "SH imploring" type.
Thus 'the SH function" of brains (ASSSs) prevents us from responding with paying/focusing distress-type actention to Specific (and by ÆPT definition always circumstantial) Hibernation Imploring Threats.
Hence, it is no less ÆPT to conclude that SH selectively switches-off consciousnessT and provides a clue to how "selective consciousness" can arise and be maintained.
Moreover, threats of "SHI type" prompt respective conditioned-in largely chronic aftermaths concEPTualised as CURSES. There already exist labels for approximately the very same subset of states of implicit memory that CURSES refers, but these are of course not as ÆPT and more or less accEPTable.
The least accEPTable of these is "engram/s"; or, rather, it is unaccEPTable for reason of being an utter euphemism and for being in use by a financially and politically shrewdly and successfully operated sect based on books written by Ron. L. Hubbard.
Another one is the almost accEPTable concepts of "primal pain" present/presented in books written by the late Arthur Janov. However, I personally feel it is an overly poignant pointer to that CURSES are incurred by lifetime challenges of SHI-type. Janov did not create a correspondingly distinct concept for what causes "primal pain" but he nonetheless came up with the Primal Theoretical notion of "primal scenes". Instead he used the most conventional tacky Freudian concept of "trauma" or "traumatic memories" (the tackiness of which can only be improved by putting it as "implicit memories of traumatising threats".
The concEPT of "SH" ties in nicely—more nicely than does "threats of SH imploring type"—with the simple and crudely schematic ÆPT definition of consciousnessT. Namely, that every different kind or content of consciousnessT is an evolved pattern of activity in 'brainspacetime' that essentially involve a neural metabolism dependent firing activity of sensory and non-sensory RAT-neurons; but of course not RAT-neuronal activity alone but complemented and more or less refined by firing of specific (S-)type sensory neurons; all of which got packaged in/by our phylogeny and ontogeny into a variety of different and in many cases mutually incompatible (and competing via means of mutual inhibition) actention modules.
The word "hibernation" also has a didactive, comparison encouraging, role to play as a reference to the generally (as opposed to specifically) muted metabolism that characterises all seasonally dormant animals — whether they be hibernating or aestivating.
Seasonal dormancy, specifically hibernation or aestivation, is a response to a seasonally/regularly frozen over or hot and dry habitat that involves preparing for and entering into a metabolically muted state that largely precludes behaviors and "actentions" (≅consciousnessT).
However, the primary ÆPT meaning of Specific Hibernation (SH) is a synaptic relay-blocking function (synaptic inhibition) that achieves a state wherein the directly affected postsynaptic neuron and neurons located farther downstream are kept 'quiet' and their metabolism correspondingly muted.
When individuals of some animal species (or subspecies) instinctively prepare for and enter into a seasonally dormant (metabolically muted) state the result is a correspondingly timed period of largely "cancelled consciousnessT" ≅ "absence of focused/paid actentions".
In contrast, when animals (e.g. we humans) cope by way of Specific Hibernation (SH) with a "threat of SH imploring type" it is mainly the ability to pay distress-type actention to this particular threat that is removed (by the SH type response).
Conversely, animals who survive and keep on coping with the automatically conditioned-in aftermath of a SHI threat can carry on trying to meet environmental (circumstantial) challenges of Opportunity type;
In other words, when we cope with a CURSES (incurred from or caused by a SH imploring threat) we remain relatively free to try to take (or to exploit) various kinds of available (environmentally present) types of opportunities (more or less directly procreation promoting such).
However, what is almost completely unique about us humans (compared to all other animals) is that we (with our exceptionally EAVASIVE neural actention selection serving systems) are this far often enough successfully (this far on the whole adaptively) 'siphon-off' neural motivational fuel generated from excitatory neurons upstream of where meassage-relaying blocking SH-type functures have been automatically deployed and are chronically working (doing so more or less efficiently/tirelessly).
These "upstream neurons" (constituents of an actention module) from which neural motivational fuel is often adaptively siphoned (rerouted), are neurons whose default function is to represent threats and be drivers of painful focuses/painfully paid (in the currency of neurometabolic resources) actentions — i.e. actentions of distress type.
[Apropos which, a CURSES, the primarily insidious "post-traumatic" (post-SHIT) aftermath of any genuine SHI type challenge, is a kind "implicit memory".]
'SH can be a very precisely localised 'synaptic relay-"gating" (and thus defensively adaptive) function. It is a function that inertly (most typically so) keeps a to distress ontogenetically dedicated actention module from temporarily becoming a mal-adaptively dominant actention module (a content of consciousnessT of an individual with a brain or ASSS).
The "presynaptic portion" of an actention module phylogenetically and ontogenetically specialised to manifest a distress-type actention, is what can come to constitute the by SH more or less sequestered excitatory core of a CURSES.
If a SH-maintaining function (or combination of such functions) brakes down or becomes itself inhibited, then the result would in most (but not all) lifetime circumstances manifest as anything from some kind of maladaptive distress-type actention, a suicide, or some symptom deserving of being labeled psychotic. A lesser 'leakage' might manifest as mild anxiety.
No leakage but instead a uniquely adaptive siphoning-off or rerouting of the excitatory portion of (excitatory neural messages generated by) a CURSES (usually more than one) will typically manifest as some more or less socially accepted or approved of "EAVASIVE" symptom.