SH justified

Specific Hibernation (SH) is the result of a pragmatic violation of the standard lexical meaning of the word "hibernation". This violation can be justified because it contributes an explanatory logic that is far more in-depth than the fact that "hibernation" contributes an H whereby to form a deservedly (and 'distinKtly') allusive acronym from the expressions: "SH imploring threats" or "perils or ordeals of SHI-type". 

It is ÆPT and advisable to didactically (autodidactically) compare SH with all examples of with seasonal regularity instinctively prepared for and entered into states of dormancy (that is aestivation as well as hibernation). Such adaptive ways of coping with seasonally harsh climatic conditions may be referred to as General 'Hibernation' (GH); this in order to keep some consistency in the somewhat twisted choice of the word "hibernation".


So, "specific hibernation" got (happened) to be ÆPTly chosen because it fitted both a MAD-inspired acronym-building aim and because the expression provides an apt reference to the obvious fact that a relatively metabolically muted metabolism and a compromised (cancelled) consciousnessT is the common denominator between SH and GH. 

Specific Hibernation (SH) refers specifically to presynaptic and/or postsynaptic inhibition that maintains an absence of firing or a sufficiently low frequency of firing, matched by a correspondingly muted metabolism, in the postsynaptic neurons and in farther downstream neural constituents of an actention module recruited by and specific to a by the sensory part of the module detected and registered (but by the SH-function prevented from becoming a "paid distress-type actention to) Specific (and by ÆPT definition always circumstantial) Hibernation imploring threat.

Hence it is ÆPT to conclude that SH selectively switches-off consciousnessT.


Moreover, threats of "SHI type" prompt respective conditioned-in largely chronic aftermaths in the form of CURSES (more or less accEPTable alternative labels are engrams (least accEPTable), primal pain, and maintain, a corresponding "selective unconsciousness". 

[SH does so as explained by the simple and crudely schematic ÆPT definition of consciousnessT. That is, every different kind or content of consciousnessT is produced by neural metabolism dependent firing activity of sensory and non-sensory RAT-neurons complemented, refined and regulated by firing of specific (S-)type sensory neurons packaged (in/by our phylogeny) into a variety of different and in many cases mutually incompatible and via means of mutual inhibition competing actention modules.]

The word "hibernation" also has a didactive, comparison encouraging, role to play as a reference to the generally (as opposed to specifically) muted metabolism that characterises all seasonally dormant animals — whether they be hibernating or aestivating.

Seasonal dormancy, specifically hibernation or aestivation, is a response to a seasonally/regularly frozen over or hot and dry habitat that involves preparing for and entering into a metabolically muted state that largely precludes behaviors and "actentions" (≅consciousnessT).

However, the primary ÆPT meaning of Specific Hibernation (SH) is a synaptic relay-blocking function (synaptic inhibition) that achieves a state wherein the directly affected postsynaptic neuron and neurons located farther downstream are kept 'quiet' and their metabolism correspondingly muted.

When individuals of some animal species (or subspecies) instinctively prepare for and enter into a seasonally dormant (metabolically muted) state the result is a correspondingly timed period of largely "cancelled consciousnessT" ≅ "absence of focused/paid actentions". 


In contrast, when animals (e.g. we humans) cope by way of Specific Hibernation (SH) with a "threat of SH imploring type" it is mainly the ability to pay distress-type actention to this particular threat that is removed (by the SH type response). 

Conversely, animals who survive and keep on coping with the automatically conditioned-in aftermath of a SHI threat can carry on trying to meet environmental (circumstantial) challenges of Opportunity type; 

In other words, when we cope with a CURSES (incurred from or caused by a SH imploring threat) we remain relatively free to try to take (or to exploit) various kinds of available (environmentally present) types of opportunities (more or less directly procreation promoting such).

However, what is almost completely unique about us humans (compared to all other animals) is that we (with our exceptionally EAVASIVE neural actention selection serving systems) are this far often enough successfully (this far on the whole adaptively) 'siphon-off' neural motivational fuel generated from excitatory neurons upstream of where meassage-relaying blocking SH-type functures have been automatically deployed and are chronically working (doing so more or less efficiently/tirelessly). 

These "upstream neurons" (constituents of an actention module) from which neural motivational fuel is often adaptively siphoned (rerouted), are neurons whose default function is to represent threats and be drivers of painful focuses/painfully paid (in the currency of neurometabolic resources) actentions — i.e. actentions of distress type.  

[Apropos which, a CURSES, the primarily insidiou"post-traumatic" (post-SHIT) aftermath of any genuine SHI type challenge, is a kind "implicit memory".]

'SH can be a very precisely localised 'synaptic relay-"gating" (and thus defensively adaptive) function. It is a function that inertly (most typically so) keeps a to distress ontogenetically dedicated actention module from temporarily becoming a mal-adaptively dominant actention module (a content of consciousnessT of an individual with a brain or ASSS). 

The "presynaptic portion" of an actention module phylogenetically and ontogenetically specialised to manifest a distress-type actention, is what can come to constitute the by SH more or less sequestered excitatory core of a CURSES.

If a SH-maintaining function (or combination of such functions) brakes down or becomes itself inhibited, then the result would in most (but not all) lifetime circumstances manifest as anything from some kind of maladaptive distress-type actention, a suicide, or some symptom deserving of being labeled psychotic. A lesser 'leakage' might manifest as mild anxiety. 


No leakage but instead a uniquely adaptive siphoning-off or rerouting of the excitatory portion of (excitatory neural messages generated by) a CURSES (usually more than one) will typically manifest as some more or less socially accepted or approved of "EAVASIVE" symptom.